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Evidence-based· Peer-reviewed research
Actionable· Improves with lifestyle
Lipid Markers

Non-HDL Cholesterol

Everything that can clog your arteries — in one simple number. Superior to LDL for cardiovascular risk prediction.

2026-018 min read

Formula

Total Cholesterol − HDL Cholesterol

Captures ALL atherogenic lipoproteins including LDL, VLDL, IDL, Lp(a), and remnant particles.

Who is this especially useful for?

  • People with elevated triglycerides (>150 mg/dL)
  • Those with type 2 diabetes or prediabetes
  • Anyone with metabolic syndrome
  • Those with family history of heart disease
  • People whose LDL is at goal but still have cardiovascular risk
  • Anyone following a ketogenic or low-carb diet

Non-HDL captures ALL atherogenic particles — everything LDL misses.

What is Non-HDL Cholesterol?

Non-HDL cholesterol is calculated by subtracting your HDL ("good") cholesterol from your total cholesterol. The result represents ALL atherogenic (artery-clogging) lipoproteins in your blood — including LDL, VLDL, IDL, lipoprotein(a), and remnant particles.

Many cardiovascular researchers consider Non-HDL a superior predictor of heart disease risk compared to LDL alone. The 2018 ACC/AHA guidelines emphasize non-HDL cholesterol, especially for patients with elevated triglycerides, diabetes, or metabolic syndrome.

Example Calculation

200

Total Cholesterol (mg/dL)

55

HDL Cholesterol (mg/dL)

=

145 mg/dL

200 − 55 = 145 mg/dL — This includes ALL atherogenic particles (LDL + VLDL + remnants).

More Complete Than LDL

LDL-C misses VLDL, IDL, Lp(a), and remnant cholesterol. Non-HDL includes everything that can deposit in artery walls. This is why it's becoming the preferred marker in modern cardiology.

Why Non-HDL Matters More Than LDL

Calculate Your Non-HDL

Uses values from any standard lipid panel.

Three Perspectives on Non-HDL

Different health paradigms interpret Non-HDL thresholds differently:

Non-HDL Interpretation by Paradigm

Standard Medical
Research Consensus
Metabolic Optimization
82
100
150
200
212.5
130160190
100130160
130180
Typical
Context-Dependent
Investigate

Standard Medical

High> 190
Borderline160 – 190
Near Optimal130 – 160
Optimal< 130

Research Consensus

Elevated> 160
Borderline130 – 160
Good100 – 130
Optimal< 100

Metabolic Optimization

Investigate> 180
Context-Dependent130 – 180
Typical< 130

Standard Medical

ATP III/ACC guidelines. Non-HDL targets are LDL goal + 30 mg/dL.

< 130 mg/dL for most adults; < 100 mg/dL for high-risk patients.

Research Consensus

Preventive cardiology outcome studies. Lower is generally better.

Target < 100 mg/dL; < 70 mg/dL for primary prevention in some studies.

Metabolic Optimization

Low-carb/ketogenic context. LDL may rise but VLDL drops.

Stable/improving Non-HDL with excellent TG/HDL suggests good adaptation.

How to Test

1
Total Cholesterol: Included in all standard lipid panels.
2
HDL Cholesterol: Included in all standard lipid panels.
3
No Fasting Needed: Unlike LDL calculations, Non-HDL is accurate in the fed state.

💡 Pro tip: Non-HDL is automatically calculated if you have total cholesterol and HDL. No separate test needed.

How to Lower Non-HDL Cholesterol

Since Non-HDL captures multiple lipoprotein types, improvement strategies are multifaceted:

Lower Triglycerides

Reduce refined carbohydrates

Major driver of VLDL production

Limit sugar and fructose

Directly raise triglycerides

Consider omega-3s

Fish oil significantly lowers TG

Limit alcohol

Significant impact on triglycerides

Dietary Patterns

Mediterranean diet

Proven cardiovascular benefits

Low-carbohydrate diets

Particularly effective for high TG

Reduce processed foods

Improve overall lipid profile

Lifestyle & Medical

Regular exercise

Lowers TG, raises HDL

Weight management

Reduces VLDL production

Address insulin resistance

Root cause of VLDL overproduction

For Keto/Low-Carb Dieters

Keto dieters may see LDL rise, causing concern. Non-HDL provides context — if triglycerides drop significantly, the rise in LDL may be offset by lower VLDL, keeping Non-HDL stable or improved. Track both TG/HDL ratio and Non-HDL for the complete picture.

Frequently Asked Questions

LDL-C only measures cholesterol in LDL particles. Non-HDL captures ALL atherogenic particles — LDL, VLDL, IDL, Lp(a), and remnants. In people with elevated triglycerides (common in metabolic syndrome), LDL significantly underestimates true atherogenic burden while Non-HDL remains accurate.
No, that's one of its advantages. Non-HDL is accurate whether or not you've fasted. LDL calculated by the Friedewald equation becomes inaccurate with elevated triglycerides or non-fasting samples. Non-HDL doesn't have this limitation.
Standard guidelines recommend below 130 mg/dL for most adults, below 100 mg/dL for high-risk patients. Research-focused targets aim for below 100 mg/dL for primary prevention. Your optimal target depends on your overall cardiovascular risk profile.
Keto dieters may see LDL rise, but Non-HDL often stays stable or improves. This is because triglycerides (and thus VLDL) drop significantly, offsetting any LDL increase. Track both TG/HDL ratio and Non-HDL for the complete picture.
Not exactly, but they're related. ApoB measures the number of atherogenic particles, while Non-HDL measures the cholesterol they carry. Both are superior to LDL alone. ApoB requires a separate test; Non-HDL uses standard lipid panel values.
Historically, LDL has been the focus of guidelines and statin trials. However, major guidelines (ACC/AHA 2018) now emphasize Non-HDL, especially for patients with elevated triglycerides, diabetes, or metabolic syndrome. Awareness is growing.

Key Takeaways

  • 1Non-HDL = Total Cholesterol − HDL Cholesterol
  • 2Captures **ALL atherogenic particles** (LDL + VLDL + IDL + Lp(a) + remnants)
  • 3Superior to LDL alone for cardiovascular risk prediction
  • 4**No fasting required** — accurate in fed state
  • 5Target below 130 mg/dL for most adults; below 100 mg/dL for high-risk
  • 6Particularly valuable when triglycerides are elevated (LDL becomes inaccurate)

ALL

Atherogenic Particles

Captures LDL + VLDL + IDL + Lp(a) + remnants

ATP III Guidelines

No Fast

Required

Accurate without fasting, unlike LDL

Clinical consensus

<130

mg/dL Target

Primary target for most adults

ACC/AHA Guidelines

Evidence-Based

This calculator is based on peer-reviewed research validated across thousands of clinical studies.

View scientific references(5)

Medical Disclaimer

This article is for educational purposes only and is not intended as medical advice. Information presented is based on peer-reviewed research but should not be used for self-diagnosis. Always discuss your lab results and health concerns with a qualified healthcare provider.